1d

Maybe a littlebit late. The rheumatic fever which occurs in about 2- 3 % of patients with a sore throat induced by a special stand of beta-Haemolytic Streptococcus of the A group is influencing several tissues. The brain, which oeada to chorea huntongton like symptoms, the joints which leads to a migratory polyarthritis and signs in the skin and the subdermal tissue. These four loci will cure after inflammation nearly to restitutio ad integrum. The inflammation of rheumatic fever, similar to a autoimmune disease, is induced because genetical preposition. The antigenes of the bacteria is veryvery similar to the bodies HLa Antigenes. So if there is an inflammation induced by the special streptococcustype mentioned above, the body will also attack the own tissue. In this case clearly also the endocard, myocard and pericard can be attacked. This inflammtion leads in the case of the endocard to buiding of lesions in the contact zone of the heart valves . There the body will build up fibrin fibres. And scar tissue which will lead to a stenosis in the first order. After rheumatic fever is gone the status stays like it is and in the next inflammation with the special antigene carrying streptococcus the process proceeds and the stenosis grows. But also the formerly even valves endocard is getzing rough. That is where the infective endocarditis is taking place. The roughness gives the bacteria a better place to grow and the turbulence behind the valve makes it even easier for them to hold on the endocard. So two factors connect the rheumatic with the infective endocarditis. A thrombkting vegetation can lead to a apoplex. BUT there are the duke criteria which have to be fulfilled.

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Nice job!

My son's mother is in the hospital also, I see you posted this 2 years ago. I pray to God that your friend is in great health now!