After watching I felt guilty, I have been working as an RT for 7 years, this is new to me. WOW BIG THANKS.

This is VERY IMPORTANT whether you're a student or not!!!! This concept is very commonly forgotten among comfortably working practitioners.

YES! It's truly the combination of our Hgb and SaO2/SpO2 that is ultimately determining our end perfusion. Vital for everyone to understand this. Thanks for explaining it so well!

Game changer! Reading title, thinking Basic, I don’t need it. You bring me back to planet earth😆you have dissected hypoxemia & Hypoxia in a way that I will not look at an ABG the same! Thankyou for getting the average out of us and producing the kind of RRT’s that we look up to. 💛THANKYOU!

It not only benefits the RT students but you have helped others as well! I teach nursing students and this helped me to understand this confidently enough so that I can teach them effectively - Thank you!

Thank you I'm learning about this now in RT class and this was a very clear and thorough explanation

I just wandered onto your video as I'm searching for information on what is happening to my son. This is fascinating. My son is an Army Infantryman. He is in great shape. He maxes out every category on his ACFT except the run. He's very good at short sprints but when he runs long distances his feet and hands start to go numb. He feels like a weight is setting on his chest, and he feels like he has a lump in his throat. He sweats profusely, feels completely wiped out, his heart rate gets to about 205 every time he runs. His buddies say he goes white as a sheet and his lips start to turn a bluish color. He has been going to medical for some time now in an effort to discover what is going on. They have done an MRI, CT Scan, lung tests, all kinds of heart tests, tested him for EIB, and other things. Long story short is they still can't quite discover what is happening. Just recently his Dr. said that he may have GERD. He has no symptoms of GERD in daily life, but they went ahead and gave him a prescription for it. He has been taking it about a week now and yesterday when he went on his daily run his heart rate only got to 175 and he felt so much better. Not great but much improved. I just began searching the web and ran across a number of articles about metabolic acidosis and hypoxia. That led me to these videos. I'm definitely going to have my son check them out. Who knows, he may even be able to talk with the Army Drs. and get them to look in this direction for some answers. :)

U r an excellent teacher!!!

Thank you! This is GOLD from beginning to end.

Excellent and very informative I wish you best of luck

g.o.a.t OF rt'S this concept is very important and appreciate you for explaining this and bringing it up again!

Love your content. Im an SRNA. Thank you.

Ur JUST AWESOME!! PLS KEEP EM COMING

in semester 2 of RT school and you DEFINITELY have prepared for my exams!! thank you so much!!

Fabulous, I will make sure to check all my patient’s hgb levels. Makes so much sense!

So many dots connected. Thx coach

Great video but I feel like the formula D02 is even more appropriate because if the patients cardiac output is low the oxygen will also not be delivered to the tissues well even if the hemoglobin is fine

Your Amazing your videos have been helping me understand everything so much better than in class Thank You

makes a lot of sense.Thank you.

Can Empyema, ( extreme), lung scraped and all cause this? I need answers, they can’t even figure that out, then I was diagnosed with this, always in the hospital. Long term. Also, can Auto Immune diseases be another situational add on? I’m absolutely going to keep this fight up!! Last month I told the care, I WILL get through this too!

I learn a lot with your presentations. Thanks.

Great

You nailed it! This is what all is going on, this is it! I even documented .

You are a life saver! 👏🏻 Good job!

all make sense now thank you

Thanks for the review!

excellant

Very informative video! Thanks a lot! However, I feel in most public hospitals, unless the hb is low to a certain level, they re reluctant to give blood tranfusion.

Great explanation.

I have ILD caused by Hypersensitivity Pneumanitis (to formaldehyde) diagnosed by Mayo Clinic after an open lung biopsy. I also have HP of 30 diagnosed by RHC which also revealed an enlarged right Ventricle & right Atrium with tricuspid 'leak'. I was given first 6 months to live then 6 year after the cause (carpeting) was removed. In order to improve the quality of my life, I transitioned my muscles to ketones (using the strict ketogenic diet for one year). Now, 20 years later, I am in decline. I arterial saturation (using finger pulseoxymeter) shows 02 sat at about 94. However, now I am showing signs of hypoxia...particularly cerebral hypoxis. (I blacked out and fell with no warning. At 77, I am lucky I did not break any bones...just bruised a lot.) How could I have hypoxia? MSM

thank you... so much point get on this video..

You Do have any video talking abaut the mexanic ventilación y acidosis stated. My english is no fine. A like hear your anf see your videos. From Nicaragua.

Thank you so much ....

Thanks doc, now i understand

I like your presentations just one request the white board is reflecting an image of a ?TV screen Thanks

Do you have a calculation to know how much you should increase your FiO2 to get a more normal PO2 with a Hgb of 11.2?

how can we improve an hypoxic pt with metabolic acidosis?

You are amazing thank you so much

hello, I have a question for the last example you gave us. The one with the CaO2 value at 21.07. Would that patient be considered to have Respiratory Acidosis? Because, for the example with the CaO2 value of 11.06, I can see how the patient would have Metabolic Acidosis; giving that the lungs work well but poor O2 transfer to the tissue due to the lack of Hb. Anything would really help, thank you.

Is it beneficial for CHF patients to test their hb before deciding whether or not to put them on a CPAP?

Clarify for me. Newbie here. So even though the pt is oxygenating at the tissue level, would we still try to oxygenate more at the external level since that is what is being produced lung wise? Everything else makes absolute sense!

What could hypothetically happen to a patient if bicarbonate, say 3 grams a day, so 1 gram every 8 hours, what could that do to them if they already have hypoxemia, in someone who is already compensating with an increased hemoglobin? Thank you.

Hey Coach, is there a way to calculate this with VBG?

Some hospital are lazy!