Dr. Seyfried refers to highly processed foods, such as packaged snacks, packaged bread, processed meats, frozen or ready meals, etc. These tend to have added sugar, refined carbohydrates, and artificial ingredients, such as preservatives.

Please remember the following: Our distinguished guest, Kristina, is not providing any health or diet advice. She is drawing attention to known cancer risks, such as microwaving food in plastic containers and consuming foods that contain nitrates, a potentially cancer-causing compound. This is an important message that needs to be shared.

@@TargetCancer Telling people what they should or shouldn't put in their body is a form of health advice. Did you know that obesity is a cause of cancer?

Correct! Thank you for your comment!

You may try to reach Dr. Seyfried through Boston College.

Please reach out to Anixa Biosciences. Visit their website www.anixa.com/

Wont make a difference. Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells.

@@hveragerthi9642 so all what dr seyfriend claims is not true you say

@@hveragerthi9642 so what dr Seyfried says is not true ?

@@clemenscohen7318 What do you think? Ketones and lactate “fuel” tumor growth and metastasis www.ncbi.nlm.nih.gov/pmc/articles/PMC3047616/ "Ketones are a “super-fuel” for mitochondria, producing more energy than lactate and simultaneously decreasing oxygen consumption.15-17" "So, just as ketones are a “super-fuel” under conditions of ischemia in the heart and in the brain, they could fulfill a similar function during tumorigenesis, as the hypoxic tumor exceeds its blood supply. Stromal ketone production could obviate the need for tumor angiogenesis. Once ketones are produced and released from stromal cells, they could then be re-utilized by epithelial cancer cells, where they could directly enter the TCA cycle, just like lactate. In this sense, ketones are a more powerful mitochondrial fuel, as compared with lactate." "Similarly, acute fasting in rodent animal models is also sufficient to dramatically increase tumor growth.22 Both of these experimental conditions (diabetes and fasting/starvation) are known to be highly ketogenic and, thus, are consistent with our current hypothesis that ketone production fuels tumor growth. Finally, given our current findings that ketones increase tumor growth, cancer patients and their dieticians may want to re-consider the use of a “ketogenic diet” as a form of anti-cancer therapy." Ketone body utilization drives tumor growth and metastasis www.ncbi.nlm.nih.gov/pmc/articles/PMC3507492/ "Thus, the tumor stroma may serve as a reservoir for ketone body production, while cancer cells upregulate the enzymes required for ketone body re-utilization, driving oxidative mitochondrial metabolism (OXPHOS) in epithelial cancer cells (Fig. 9). To prevent this form of “two-compartment tumor metabolism,” ketone inhibitors should be designed to halt ketone body production in cancer-associated fibroblasts and ketone body re-utilization in epithelial cancer cells. This simple strategy could effectively starve cancer cells to death by “cutting off their fuel supply.” Finally, it is worth noting that ketogenic fibroblasts were more prone to a loss of stromal Cav-1 expression. In breast cancer patients, a loss of stromal Cav-1 expression is associated with increased tumor recurrence, metastasis, drug resistance and overall poor clinical outcome.10-13 Thus, stromal Cav-1 could be used as a biomarker to select patients that would be more likely to benefit from therapy with ketone inhibitors, allowing biomarker-based treatment stratification and personalized cancer therapy." Ketone bodies and two-compartment tumor metabolism www.ncbi.nlm.nih.gov/pmc/articles/PMC3507491/ "We have previously suggested that ketone body metabolism is critical for tumor progression and metastasis. Here, using a co-culture system employing human breast cancer cells (MCF7) and hTERT-immortalized fibroblasts, we provide new evidence to directly support this hypothesis. More specifically, we show that the enzymes required for ketone body production are highly upregulated within cancer-associated fibroblasts. This appears to be mechanistically controlled by the stromal expression of caveolin-1 (Cav-1) and/or serum starvation. In addition, treatment with ketone bodies (such as 3-hydroxy-butyrate, and/or butanediol) is sufficient to drive mitochondrial biogenesis in human breast cancer cells. This observation was also validated by unbiased proteomic analysis. Interestingly, an MCT1 inhibitor was sufficient to block the onset of mitochondrial biogenesis in human breast cancer cells, suggesting a possible avenue for anticancer therapy. Finally, using human breast cancer tumor samples, we directly confirmed that the enzymes associated with ketone body production (HMGCS2, HMGCL and BDH1) were preferentially expressed in the tumor stroma. Conversely, enzymes associated with ketone re-utilization (ACAT1) and mitochondrial biogenesis (HSP60) were selectively associated with the epithelial tumor cell compartment. Our current findings are consistent with the "two-compartment tumor metabolism" model. Furthermore, they suggest that we should target ketone body metabolism as a new area for drug discovery, for the prevention and treatment of human cancers."

Studies have shown that ketones are not only a fuel source for cancer cells, but they also promote metastases as the ketones activate hyaluronidase and proteolytic enzymes needed for natural metastases.

Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells. Look in to ozone therapy, which addresses cancer through a variety of mechanisms, including selectively destroying cancer cells in a chain reaction through a peroxide overload.

Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells. Look in to ozone therapy, which addresses cancer through a variety of mechanisms, including selectively destroying cancer cells in a chain reaction through a peroxide overload.

Phthalates are a class of chemicals that are used for many purposes: to retain the scents used in fragrances, to soften and strengthen plastic, and to help topical products like lotions and cosmetics stick to and penetrate skin.

Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells. And yes, glucose is generated by the body from a variety of sources through a process known as gluconeogenesis. It is also true that the body's pH levels are tightly regulated primarily by respiration and kidney function. Even a slight shift out of normal, high or low, and the pH is brought back in to normal range rapidly in most cases. Our pH is not regulated by what we eat or drink.

@@hveragerthi9642 Thank you for answering much appreciated.How can they miss this obvious fact?I still hope we are missing something and someone will explain as i would like to starve the cancer but even on keto when measured blood glucose levels never go to 0 so cancer always gets energy and it can grow.

@@lacipeter6934 There is more misinformation spread on the internet and in books than any other other disease or condition. Often the misinformation gets spread because someone is trying to sell a cancer product or a book. So they simply say what they think people want to hear, or repeat whatever nonsense they read elsewhere without fact checking the science because it fits their needs. Some commonly spread cancer myths include: -Cancer can be starved -Cancer cells arise in acidity and are killed by alkalinity -Cancer cells cannot survive in a highly oxygenated environment -That sugar causes cancer -That a ketogenic diet cures cancer -That you can alkalize the blood to cure cancer Worse yet, when people want to convince others that what they are claiming is supposedly true, they just tack on that Nobel Prize winner Otto Warburg made the claim, which almost always is not the case. The second most common thing people get misinformed about concerning health is the alkalizing cures disease nonsense. If we become more alkaline than normal, the person can become very ill. Sightly further off and the person ends up in a coma. Sightly more alkaline and the person will die from lack of oxygen. This is one of the reason that our bodies regulate our pH so tightly. Cancer cells also arise in alkalinity all the time, and they have an alkaline internal pH that allows them to survive and drives cancer cell glycolysis. Studies have also shown that when healthy cells are made excessively alkaline, the healthy cells morph in to cancer cells. Most pathogens also thrive in alkalinity, and are killed by acidity. This is one of the roles of stomach acid, to kill ingested pathogens. And why some pathogens use the enzyme urease to form highly alkaline ammonia to help them survive.

I lowered my dogs carbs with canned food & added fenbenezole & melatonin ( a strong antioxidant) & she’s been cancer free since 2021

@@sharonhoffman4588 wow thats good news.What type of cancer? And how long until it was gone?Any other meds?

Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells. Ketones also promote metastases by activating hyaluronidase and proteolytic enzymes needed for natural cancer cell metastases.

@hveragerthi9642 so, the keto diet does not help cure cancer?

@@angelikamarjanovic837 I have not seen any real evidence that it cures cancer. Just unfounded claims, which are common with cancer. There is more nonsense posted about cancer than any other disease or condition known.

@@angelikamarjanovic837 Read the research: Ketones and lactate “fuel” tumor growth and metastasis www.ncbi.nlm.nih.gov/pmc/articles/PMC3047616/ "Ketones are a “super-fuel” for mitochondria, producing more energy than lactate and simultaneously decreasing oxygen consumption.15-17" "So, just as ketones are a “super-fuel” under conditions of ischemia in the heart and in the brain, they could fulfill a similar function during tumorigenesis, as the hypoxic tumor exceeds its blood supply. Stromal ketone production could obviate the need for tumor angiogenesis. Once ketones are produced and released from stromal cells, they could then be re-utilized by epithelial cancer cells, where they could directly enter the TCA cycle, just like lactate. In this sense, ketones are a more powerful mitochondrial fuel, as compared with lactate." "Similarly, acute fasting in rodent animal models is also sufficient to dramatically increase tumor growth.22 Both of these experimental conditions (diabetes and fasting/starvation) are known to be highly ketogenic and, thus, are consistent with our current hypothesis that ketone production fuels tumor growth. Finally, given our current findings that ketones increase tumor growth, cancer patients and their dieticians may want to re-consider the use of a “ketogenic diet” as a form of anti-cancer therapy." Ketone body utilization drives tumor growth and metastasis www.ncbi.nlm.nih.gov/pmc/articles/PMC3507492/ "Thus, the tumor stroma may serve as a reservoir for ketone body production, while cancer cells upregulate the enzymes required for ketone body re-utilization, driving oxidative mitochondrial metabolism (OXPHOS) in epithelial cancer cells (Fig. 9). To prevent this form of “two-compartment tumor metabolism,” ketone inhibitors should be designed to halt ketone body production in cancer-associated fibroblasts and ketone body re-utilization in epithelial cancer cells. This simple strategy could effectively starve cancer cells to death by “cutting off their fuel supply.” Finally, it is worth noting that ketogenic fibroblasts were more prone to a loss of stromal Cav-1 expression. In breast cancer patients, a loss of stromal Cav-1 expression is associated with increased tumor recurrence, metastasis, drug resistance and overall poor clinical outcome.10-13 Thus, stromal Cav-1 could be used as a biomarker to select patients that would be more likely to benefit from therapy with ketone inhibitors, allowing biomarker-based treatment stratification and personalized cancer therapy." Ketone bodies and two-compartment tumor metabolism www.ncbi.nlm.nih.gov/pmc/articles/PMC3507491/ "We have previously suggested that ketone body metabolism is critical for tumor progression and metastasis. Here, using a co-culture system employing human breast cancer cells (MCF7) and hTERT-immortalized fibroblasts, we provide new evidence to directly support this hypothesis. More specifically, we show that the enzymes required for ketone body production are highly upregulated within cancer-associated fibroblasts. This appears to be mechanistically controlled by the stromal expression of caveolin-1 (Cav-1) and/or serum starvation. In addition, treatment with ketone bodies (such as 3-hydroxy-butyrate, and/or butanediol) is sufficient to drive mitochondrial biogenesis in human breast cancer cells. This observation was also validated by unbiased proteomic analysis. Interestingly, an MCT1 inhibitor was sufficient to block the onset of mitochondrial biogenesis in human breast cancer cells, suggesting a possible avenue for anticancer therapy. Finally, using human breast cancer tumor samples, we directly confirmed that the enzymes associated with ketone body production (HMGCS2, HMGCL and BDH1) were preferentially expressed in the tumor stroma. Conversely, enzymes associated with ketone re-utilization (ACAT1) and mitochondrial biogenesis (HSP60) were selectively associated with the epithelial tumor cell compartment. Our current findings are consistent with the "two-compartment tumor metabolism" model. Furthermore, they suggest that we should target ketone body metabolism as a new area for drug discovery, for the prevention and treatment of human cancers."

Thank you for your comment!

In layman's terms, what type of Cancer did you have or have?

@@traceyrychewhite1874 breast cancer

Sebi was a quack who was charged with practicing medicine without a license as he was never a medical doctor. Contrary to the propaganda, he lost the case when he took a plea deal to avoid prison time. Read the Court transcripts like I did. He was later arrested for money laundering, and died from pneumonia in jail due to his poor health. Sebi had no idea how the body actually worked, which is why this quack falsely claimed that all diseases are due to mucus, and also pushes the bogus "alkaline diet" myth. Mucus does not cause any diseases, but can increase with some diseases as part of the immune response to help protect us in those diseases. And diet DOES NOT regulate our pH.

Studies have shown that cancer cells can use glucose, fructose, ketones, lactate, fatty acids and various amino acids as fuel sources. Since may of these are generated by the body, it is impossible to starve cancer cells.

We're glad you found the information helpful. Feel free to share the video with friends who may be interested in this information.