Yes please do a Full episode on treatment for cancer cachexia.💜🙏

Thank you. Moments on meat diet please

So what creates this damaging free radical?

So how do I reduce inflammation/ lower number of particles with nutrition? What foods can I eat?

So Dr Loh what foods, drinks cause the inflammation in the first place…please advise? Did I miss it?

So how to fix or prevent this phenomenon?

I think I’m convinced. No more alcohol 😅

Hi Hershey cocoa powder is there lead cadinum or what ever chemical please let me know thank you .

I've read that the brain likes routines, as such, you don't necessarily want to alter your sleep hours in the attempt to make up the lack of sleep.

You are my hero. You saved my life through Transform.

Really educative video. I appreciate your effort to teach us about the various harmful effects of lead and cadmium. Thank you❤❤

Could you just cut to the chase and tell us how to buy it without heavy metals since I don't want either lead or sugar.

“When you’re in a broken system, that system only knows to recreate versions of itself and will not allow you to create something new” Profound and so true.

1:01:18 so you wouldn’t recommend cannibalism?

Q: what is the optimal eating pattern for a late night shift worker (who works till 1am and sleeps around 3am)?

Damn 13:07 happens to me often when I dream i’m running away from someone chasing me or something and I wake up but my body is locked! Lol now I know why

Ok…how do we get more REM?

I’m a scholar I’ve been studying the stuff for years it’s true

I hate to say it, but everything is poison. It ain’t gonna kill you right away but when you get cancer one day, you don’t know where it came from that’s the sad news. Even the air and water is poison fluoride the air got aluminum in it phosphate and even the medicine so you think that’s a accident lol your phosphate is in the lakes and screams so everything is poison what about that?

“Listen, snake venom is natural!” 😂😂 I’m going to use that line

👍

What can humans eat?

Is there thermochemistry videos my exams is in25/6

Sweet lady and interesting information. I love dark chocolate, it's one of my favorite snacks but with this in mind I try now to lower my consumption and go for 70% cocoa instead of 85/90%.

It is sad to think that medical text books don't do a good job of updating their content in light of new research. My first exposure to that idea is that the vascular endothelium glycocalyx (yes, that YT search is what led me to your channel) is not shown in the graphics, or pictorials. Why that is isn't hard to understand because when dissection is done, the glycocalyx is gone, when slides are prepped, the glycocalyx is destroyed, it is only recently that the glycocalyx has been visualized with cameras in vivo. And there are few doctors that are on a path of continuous education, even if the text books were current. So we want to tread lightly, and don't buy into, or consume someone's theory without understanding the foundation concepts. Sometimes, when you pull back the curtain a little and look - there is no foundation. Sometimes the foundation has good information, but gets stuck with goofy names because our understanding is confined by the limitations of technology. _We see as through a glass, darkly_ I am beginning to consider the life of LipoProteins as a continuum in a process, rather than existing in our pre-defined buckets of classification. Keep learning, and thanks for sharing!

Solutions 34:01: chelation with citric acid ,calcium EDTA , pyridoxamine and benfotiamine , Make sure you are not deficient in minerals and vitamins. Iron, zinc, calcium deficiency increase heavy metal absorption.. Children under 12 and pregnant women must abstain or eat small amounts with a meal to decrease heavy metal absorption .Please read Consumer Reports article : Lead and Cadmium could be in your dark chocolate. In it they show various bars with metal percentages found. Least amounts found in : Mast , Taza 70% cacao ,Ghirardelli 86% ,72% cacao and Valrhona 85% cacao .

First. (commented 4 Al Gore's rhythm ;)

Is there really much of a difference in the cause of atherosclerosis vs. aneurisms?

My working theory is that atherosclerosis is a one-two punch, or like a binary explosive (but lots of little firecrackers). There are many compounds that can wash out the glycocalyx. The glycocalyx acts as our shields, and It takes many hours for it to be restored. Focusing on high blood glucose as a wash-out mechanism, in the Standard American Diet (SAD) we get glucose spikes from multiple meals a day (including snacks) sugary drinks (including 100% fruit juice). The glycocalyx just doesn't have a chance to properly rebuild - sleeping hours isn't enough time. That all cascades much worse with insulin resistance / diabetes. Glucose itself can glycate any cell, but is it enough to cause an inflammatory reaction? Sugar by itself has been declared to be 'safe' in the 'science'. With the glycocalyx washed out, Nitrous Oxide is not released, so blood pressure may increase. Hypertension is now seen as part of the greater metabolic disorder. Oxidized products are inflammatory, and damaged LDL is a problem - identified as small, dense LDL. As you say, damaged enough as to be not recognized by the usual receptors to be removed from circulation (so they build up) , and to be identified as a foreign body by our immune system. But the vascular walls are well protected by the glycocalyx from most of the many toxins we are exposed to except for specific high-flow sites, like branches and sharp turns. Damage is minimal. Industrially processed seed oils are highly oxidative, but are Generally Recognized As Safe (GRAS). But in the continuous presence of all of the glucose in our diet, our shields are down, and the sdLDL and other oxidized products cause injury to and overwhelm the epithelium, and make their way into the vessel wall, where the auto-immune response ensues. sorry for mangling the terminology. I see the problem as the *combination* of sugar and oxidized seed oils in our ultra-processed food diet. Separately, not so bad. Our body can handle a little sugar from fruit, milk, or honey. The seed oils, it just tries to sequester that stuff. And something I haven't seen talked about - why would there be a mechanism to wash out our vascular endothelial glycocalyx on a temporary basis? That would be the flight-or-fight mechanism, in my estimation. Cortisol + Adrenaline dump glucose, washing out the glycocalyx keeping the blood vessels restricted and increasing blood pressure with the increased blood flow to push energy to every nook and cranny of the capillaries. This runs the risk of damage for a time, during an emergency. _Desperate Times Call for Desperate Measures._ It doesn't seem like running marathons continuously are good for longevity. As for me, the best strategy to eliminate sdLDL and other factors is whole blood donation. Maybe there was something to the practice of blood-letting. Today it is a win-win situation, good for society as well.

Are you seeing the evidence from the Lean Mass Hyper-Responder (LMHR) studies? Dave Feldman, Nick Norwitz PHD, Dr. William Cromwell. I look at a LDL-cholesterol/apolipoprotein B ratio of 1.52 as good (at this time)

Well then, to borrow from others: Our shields (glycocalyx) are down long enough for inflammation to take hold (catch fire) in the endothelial layer / vascular wall. The immune system dispatches fire trucks (lipoproteins) to deliver the firefighters. Years ago, with rudimentary research tools, we found the outlines of turnout gear (cholesterol? maybe? phytosterols?) of firefighters at the scene of the conflagration, and deemed the fire trucks guilty of arson, and started impounding them (with statins).

And yet, we end up affecting cholesterol synthesis in the brain. sigh. From the medical establishments perspective I am taking a crap shoot anyway. I have minimized carbohydrate intake and removed industrially processed seed oils from my diet. Having a CAC score of 543, I am starting 1mg per day of pitavastatin for the the pleotropic effects, rather than lowering LDL. next therapy: more exercise, less YT video watching. "Eat more, move more"

Thank you so much Vyvyane! This is brilliant. I wasn't aware you could be have deficiency and excess at the same time across different organs.

Thank you so much for addressing this very important topic in such an honest, courageous, and well-spoken way!

Thank you for this video. I have a huge amount of respect for the effort you put into this, even though as a layperson, much of the science is above my understanding. I was actively seeking out critical reviews, as the skeptic in me wants to find balance on bold panacea statements. I will say that Tom Dayspring has had a massive impact on my own layperson understanding of the mechanisms of lipids and atherosclerosis, to the point that I did my own Boston Heart Cholesterol Balance test and found high levels of cholesterol absorption markers (camposterol and beta-sitosterol) and low levels of production markers (lathosterol and desmosterol), which led me to question whether ezetimibe monotherapy might be a more appropriate first line drug than a statin which targets synthesis. I now feel cognitively suspended between trusting current guidelines (statin first) vs what seems to be a different emerging view toward individualising the therapy (ezetimibe first). I’d be very interested in hearing your thoughts about tailoring treatments for hyper-absorbers vs. hyper-producers, as Tom Dayspring spent quite a lot of time differentiating between. Is this based on fuzzy science and thus the guidelines haven’t validated this, or is this a valid approach that just has not yet been adopted due to the slow pace of change. Sorry if my terminology is off - I’m just a patient interested in learning more to advocate for my own primary prevention in a family with lots of cardiovascular issues. (I am also aware of the Dunning-Kruger effect of the danger of knowing a little bit and making large sweeping assumptions, as a layperson)

Note: There is a sound glitch at 32:36 and I was saying "spinach is rich in oxalic acid". This interferes with iron absorption.

Fantastic, thank you.

Thank you for the presentation. I’m very impressed. Can you do future episodes on 1) LMHR; 2) Lp(a) and oxidized phospholipids?

Cardiac cachexia. My husband is going through it now following his last acute heart failure exacerbation. There if virtually no information about it.

Nice..would like to hear more from you

Correction: It's not the Mavik protocol, but the Marik protocol with vitamin C dosing (by Dr. Paul Marik). Kudos and thanks to Beth Richardson for reaching out with that correction!

Great work! I have enjoyed/binged a few of your videos today!

BS

Just listened to you on StemTalk. Wow! Looked you up on YT and subscribed.

Pls give solution straightforward

I am totally baffled. You put forward a thesis statement in the intorduction; a dissemination of Malcolm Kendrick's blood clot-artheriosclerosis theory.........and then never referred to him, his theory or his work again. So do you agree with him or not?

Yes- please more on cancer cachexia.. especially in disabled population. They can’t do much physical activity.. so is Whole food plant based diet best option?

Thank you for your efforts to convey highly complex subject matter to the non-medical community. Drs. Attia and Dayspring are part of a group of doctors and researchers that believe that ASCVD is a function of lifetime exposure to ApoB - and to avoid ASCVD individuals should take more aggressive steps to lower ApoB than is currently recommended by the American Heart Association (which is to treat high LDL cholesterol with lifestyle and meds (statins as first line, ezetimibe and PSCK-9 inhibitors as second line). Dr. Attia has said - you should "slam your ApoB" and by this he means get it to a level of 30 mg/dl, and do so with statins, ezetimibe and/or psck-9 inhibitors. Of course, there are side effects and risks (like increasing insulin resistance, etc.), but the subtext is that these risks are most certainly worth taking to avoid the plaque buildup in arteries. Your discussion seems to indicate the artery wall has issues that lead to susceptibility to ApoB particles causing further damage, but without that arterial wall damage, the ApoB would not have such an effect. This is not equivalent to the "limit ApoB lifetime exposure" model which seems to indicate there is just some probability of ApoB causing damage so the more of it the higher the probability and eventually you have full blown ASCVD. Can you speak to this, and would you agree that limiting ApoB is indicated based on what is known presently? Thanks,

As part of the immune cascade forming the clots, it would be interesting if science would address the more recent phenomenon of the fibrous rubbery clots forming in artery walls of both the living and cadavers - not the typical jelly clots. Is this because of prolonged inflammatory irritation from spike proteins? This is really the elephant in the room, especially in the young. Myocarditis was common. As part of a study conducted here in Texas, I am aware that mrna generated spike tended to last quite a long time in the bloodstream. Naturally occuring spike went away within a few months. Nucleocapsids from natural viral infection remained for a long time.

What you refers as micro calcification is commonly known as soft plaques, is that right? It is undetectable with CAC scans. But soft plaque may break off & form clots down stream, stroke, heart attack. Calcified plaque is more stable, so statin may raise CAC score but reduce risk. I can't take statin - bad painful cramps that go away as soon as I stop starins.