Conn's syndrome (Primary Hyperaldosteronism)

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Conn's syndrome (Primary Hyperaldosteronism) - MADE EASY

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11 жыл бұрын

Conn's syndrome vs Secondary Hyperaldosteronism
Conn’s syndrome also known as Primary Hyperaldosteronism, is a tumor of adrenal glad which causes excess production of a hormone called aldosterone. The major function of aldosterone is to reabsorb sodium and excrete potassium and acid (H+). Conn’s syndrome can be either a unilateral tumor (located on only one adrenal glad) which are also the most common adrenal tumors or bilateral tumor (located on both adrenal glands). The reason Conn’s syndrome is important is because of the fact that it is potentially curable cause of high blood pressure (hypertension).
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What is Conn's syndrome
What is the most common cause of Conn syndrome?
What symptoms are associated with Conn's syndrome?
How do you test for Conn's syndrome?
Is Conn's Syndrome serious?
Can too much aldosterone cause weight gain?
What is primary hyperaldosteronism?
What is Secondary Hyperaldosteronism?
Conn's syndrome vs Secondary Hyperaldosteronism?
Conn's syndrome and Secondary Hyperaldosteronism?
What is hypertension?
What causes hypertension?
Do I have hypertension?
Causes of hypokalemia?

Пікірлер: 61

@DavidDaley23 7 жыл бұрын

Great video. Very clearly presented and informative. Just one point to consider regarding the sodium concentration in secondary hyperaldosteronism. Due to congestive heart failure (one of the common causes of secondary hyperaldosteronism), patients often become sodium and water overloaded due to increased renin and thus aldosterone release. The increased renin occurs becuase of reduced effective intravascular volume which is sensed in the kidney as you clearly outline. The reabsorption of sodium and thus water leads to hypervolaemia which we see clinically by a raised JVP and peripheral oedema in some patients. However, angiotensin II also stimulates ADH secretion from the posterior pituitary gland as well as renin secretion from the kidney. ADH acts differently to aldosterone and results in water (but not sodium) reabsorption in the collecting duct. Therefore, although patients with heart failure for example are often sodium overloaded (due to the effect of hyperaldosteronism), the disproportionately greater reabsorption of water means that the patient actually can have low sodium concentration in the extracellular fluid (hyponatremia) due to the dilution effect of increased water intake.

@adeniyiadeaga864 Жыл бұрын

You must be a genius. Very clear explanation. Thanks

@PhayzeGee 9 жыл бұрын

Wow, you explained it so concisely-yet so simply as well. Thank you

@MatirManush-wu9cc 6 жыл бұрын

Fezile Gabellah, How are you doing ? We hope you succeeded very well.

@puglife6550 2 ай бұрын

Laid in the hospital after successful surgery. Less than 24 hours since the tumour and gland removed and already there has been significant improvement.

@Joe_Lj 7 жыл бұрын

Good vid1 One correction: s-Na+ will be normal as the Na+ reabsorption will be neutralized by the concomittant H20 reabsorption.

@drvishalpatil 6 жыл бұрын

no one can make it any more easy, thank you :)

@flex8514 7 жыл бұрын

Really helps to understand. Thanks.

@bhalchandrabhalerao 10 жыл бұрын

thank god i clicked on your link. very excited to see what you have to offer. stay fit.

@StarLyrics-qw9ll 3 ай бұрын

Short and simple bro ✌️

@reco45rs 7 жыл бұрын

Very clear and concise. Thank you!

@anshumanjha3138 6 жыл бұрын

Really well explained. Thank you and well done.

@bonnieromick9397 2 ай бұрын

Great lecture. Thank you

@marioramirez21 11 жыл бұрын

Always great lectures, thank you so much

@thepetsaver 10 жыл бұрын

fantastic video, thank you!

@edwincelsovilcapajares9775 8 жыл бұрын

you are a good teacher

@medaddict2145 10 жыл бұрын

very easy to follow and great explanation thank you

@FFOTFoltyn 3 жыл бұрын

Brilliant presentation

@TheMobster450 7 жыл бұрын

fantastic video explained very well good job

@ibrahimmi317 10 жыл бұрын

If u can.post for D.insipidus and SIADH .. Would be great !

@dr-helal2659 10 жыл бұрын

love it, thanks a lot for ur time

@parindatusmeehaque1966 Жыл бұрын

Thank you

@s.u.k.k.u8765 3 жыл бұрын

Very nice.

@edwincelsovilcapajares9775 8 жыл бұрын

Really your explication is very good. Made Easy is tru

@DailyMedEd 8 жыл бұрын

+Edwin Celso Vilca Pajares, Thank you, we're glad you found this video tutorial helpful. Please check out our other videos and feel free to share them with others :)

@sahithya1198 9 жыл бұрын

beautifully explained. thank you

@elpedro812 4 жыл бұрын

Consider teaching

@parsaamin4789 8 жыл бұрын

Great lecture!

@letsmakesomepeace 9 жыл бұрын

Awesome.. Really helpfull thanx a lot :))

@nellyhoffman6194 8 жыл бұрын

Great lecture thanks ! :)

@edris.alkozi 8 жыл бұрын

perfect explanation tnx

@DrRomikVadhvana 7 жыл бұрын

Thank you so much - wonderful seriously.

@muhammadnada9433 7 жыл бұрын

thank you so much .very helpful

@NENE279 7 жыл бұрын

in primary hyperaldosterinoism the Na will be normal dt Aldosterone escape metabolism.

@muskaaaaaan Жыл бұрын

Very nice!

@mariachalsev9219 8 жыл бұрын

I have high Aldosterone (2x-3x normal max value) and severe hypertension BUT normal renin, normal Na+ and normal K+ and no protein in my urine, no cerosis, no adrenal tumors. I've gone to many doctors and they are clueless..:/ would appreciate any ideas as to what I might have.

@raptinbyjasma891 5 жыл бұрын

Maria C check medications! These may be a contributing cause?!

@jidinharidas8639 3 жыл бұрын

How you doing right now

@amiefox2719 Жыл бұрын

I know it's been a couple years, but I have the same issues. I've now been on Spiro and clonidine patches for a long while and my BP is under control. How are you doing?

@robaibrahim966 5 жыл бұрын

THANKS!

@nilkanthasharmapoudel5700 7 жыл бұрын

very very thxu i understand your lecture ...........god bless you....

@blackhorse385 10 жыл бұрын

Thanks, very good explanation and simple, dude!

@omarabrams2941 Жыл бұрын

Hi. Can you explain what happens to the levels of angiotensin 1 and 2 in conns syndrome? Renin is obviously low due to primary hyperaldosteronism. Thanks

@zahraamaan3547 8 жыл бұрын

How can I get your rest videos plzzzzzz???????

@sushantakhomdram9740 7 жыл бұрын

what does escape phenomenon mean???

@danirofoo 7 жыл бұрын

Thank you so much

@nb9536 Жыл бұрын

Great video, what diet is recommended for patients. I would imagine, DASH diet, low sodium, high potassium. What about water intake. 😊😊😊

@puglife6550 Жыл бұрын

I have conn's. Clean diet but spironolactone retains the potassium loss. This or eplerenone is the drug of choice. Alongside controlled bloodwork to monitor potassium levels and kidney function. I nearly died from the potassium loss. I'm lucky.