Paralysis by overanalysis

That's not even a joke 😅. Stress and lack of sleep probably kill more people than small dietary deficiencies.

You have new one with him on a channel called Diary of the ceo

It's very applicable to all of science. Like Richard Feynman once said (paraphrasing here), it doesn't matter how wonderful your idea and your model is, if it disagrees with experiment then it's wrong.

He also said "imagine if the Pilgrims ate bobcat with the Indians. We'd be eating pu**y every Thanksgiving." 😂

In theory, yes, but evidence no longer supports this. The real utility of eating frequently is ultimately getting enough food in without needing inedibly large portions

That is is simply untrue and made up dude. Try to be a little bit more critical.

link?

It’s not made up.. go look it up on their website!

The FDA thing was a bit of a reach but glyphosate is actually in our foods that isn't a lie. The FDA, USDA, DOH.. one of those expensive, tax funded governing bodies have to know because I and other people know. They just let it slide on though to the supermarket shelves. The WEF is pushing _"bug based food"_ that isn't a lie either. Their thought process is it would be a solution to starvation. They are worried of potential livestock shortages in the future due to human overpopulation. I'm not sure how KZfaq handles external hyperlinks but a quick google search "WEF insects" and "Glyphosate quaker" you can see for yourself.

The FDA thing was a bit of a reach but glyphosate is really in our food supply that isn't a lie. The FDA, DOH, USDA.. one of our many expensive, highly staffed, tax payer funded, governing bodies has to be aware of this long before I was. Still these products still end up on our supermarket shelves 🤷‍♂️. The WEF is pushing for insects as food. This is very true. They believe the human population is too high and we can't supply enough meat for the entire population. This is their solution. Bug based food. I'm not sure how KZfaq handles external hyperlinks. If you google "WEF insects" and "Glyphosate Quaker" you can see for yourself.

Tell that to people who listen to Brad Stanfield or follow keto and carnivore diets.

I wonder if other macronutrients affect things as well; we know they affect autophagy at least

Any evidence-based fitness enthusiast is probably eating over 1.6g/kg of protein due to the most current evidence on muscle hypertrophy. So that’s why people in these comments care a lot.

​@@danielbrown001 If you're in your 20s or 30s and a lean healthy bodybuilder, go for it. 2.2 g/kg is likely fine, but you're not likely going to see any significant (noticeable) gains from eating more than 1.6 g/kg due to the diminishing returns of the concave-down curve of muscle gains vs protein. It's best to put your efforts into an optimal training program and recovery (yeah, the hard stuff) rather than focusing on the easy stuff of eating more than 1.6 g/kg of protein. Yes, people should care. Once you get in your 50s, fitness enthusiasts begin to think more about longevity and disease. Then it comes down to: why take the risks when you're not going to get benefit from eating over 1.6 g/kg. It's no longer about ekeing out every additional nanogram of muscle mass. There's plenty of research showing protein has a negative impact on all nine aging pathways, plus negatively influencing disease. You can gamble by believing this is all bunk, but to what end? Concerns about sarcopenia in the elderly? Research shows that the additional protein does not help in the elderly. They need to lift weights. Period. 1.2 g/kg is enough, when half are not even getting 0.8 g/kg.

@@danielbrown001 Yes, people should care. Once you get in your 50s, fitness enthusiasts begin to think more about longevity and disease. Then it comes down to: why take the risks when you're not going to get benefit from eating over 1.6 g/kg. It's no longer about ekeing out every additional nanogram of muscle mass. There's plenty of research showing protein has a negative impact on all nine aging pathways, plus negatively influencing disease. You can gamble by believing this is all bunk, but to what end? Concerns about sarcopenia in the elderly? Research shows that the additional protein does not help in the elderly. They need to lift weights. Period. 1.2 g/kg is enough, when half are not even getting 0.8 g/kg.

Depend of what *boost* we are talking about 💉💉

@@francisproulx9669the booster juice 😅

The little old people drink from the grocery store.

@@Nothingmuch1039 yeah I know it was just a silly joke on my part

@@liamturner-beauchamp1570 😂😂

Greek yogurt, beans, tuna, peanut butter, tofu, cereals are all processed. You gonna avoid all those foods?

Literally everything you eat besides raw fruit and some rare veggies that are also eaten raw, I'd processed.

Kinda Richard Simmons bro😂

You don't need to lower saturated fats. A meta analysis of all the data on the highest level of data on this compound was conducted and they wrote a paper that their was NO correlation between saturated fat and negative health effects. Literally no data in the literature proves saturated as being harmful as a standalone compound, and NO it does not cause diabetes. All it does is make you sugar intolerant which is a protective mechanism in adaption to low carb diet. So don't fear saturated fats in any amount, mainly if you're low carb.

Obviously it the protein LOL

Not necessarily, it also depends on the university, and the teaching program, plus the professor's preference.

Thomas focuses on mechanisms, Layne focuses on the end product.

Oh, you deleted your response Layne, I wonder why?

Just for the people who don't speak dutch I will translate this user's name. It says "Anus door" Don't ask me why though.

Hi, they used this type of mice since it has been shown that these mice can form plaques similar seen in human. Therefore this would help to study the outcomes in the disease context, here cardiovascular diseases. Of course even having such a mice model, it is hard to translate every finding into human trials. Hope that helped. (References: Pendse AA, Arbones-Mainar JM, Johnson LA, Altenburg MK, Maeda N. Apolipoprotein E knock-out and knock-in mice: atherosclerosis, metabolic syndrome, and beyond. J Lipid Res. 2009 Apr;50 Suppl(Suppl):S178-82. doi: 10.1194/jlr.R800070-JLR200. Epub 2008 Dec 5. PMID: 19060252; PMCID: PMC2674752. Lo Sasso G, Schlage WK, Boué S, Veljkovic E, Peitsch MC, Hoeng J. The Apoe(-/-) mouse model: a suitable model to study cardiovascular and respiratory diseases in the context of cigarette smoke exposure and harm reduction. J Transl Med. 2016 May 20;14(1):146. doi: 10.1186/s12967-016-0901-1. PMID: 27207171; PMCID: PMC4875735)